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In contrast to inhibiting H 2 S biosynthesis, some studies have addressed the advantage of poisoning cancer cells with high levels of H 2 S, by using donors that release H 2 S in the tumour and somehow block the most important signalling pathways sustaining cancer survival, such as PI3K and MAPK

The results showed that SSPH-MPN and SSPH-PM had similar levels of AST, ALT, CRE, and BUN, all of which were lower than those in the saline-treated model group (Fig

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