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For CMS, the currently identified potential pathogenic mechanisms only show a negative correlation with CMS, lacking direct evidence of causality
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But the SEAL trainees using peptides were not too worried about getting caught
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NO can react with the cysteine of GSH to form GSNO that may be considered an endogenous NO reservoir, which can release it when it reacts with Cu + or with denitrosylating enzymes such as Trx and GSNO reductase (Sengupta and Holmgren, 2013)
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