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This does not constitute an absolute contraindication, but reflects the need for cardiovascular stabilisation before introducing new medications

The key distinction is that MGF biology comes from endogenous IGF-1 gene splicing and E-peptide research, while PEG-MGF is an exogenous modified compound discussed mostly in investigational and non-approved contexts [3] [10]

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Four distinct patient populations gravitate toward sub-therapeutic doses, each with a different clinical rationale

These receptors have been extensively investigated in models of neuropathic pain, migraine, and irritable bowel syndrome (Srinivasan et al

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