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Protective effect of N -acetyl-l-cysteine on amyloid [beta]-peptide-induced learning and memory deficits in mice
Researchers are still figuring out exactly how this works, but the most likely hypothesis at this point is that these effects are mediated by an increase in blood flow to the muscles, which reduces oxidative stress and improves cellular signalling related to muscle recovery
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Niger J Physiol Sci
Expression profile and prognostic value of NNMT in patients with pancreatic cancer
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