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BPC-157's angiogenic effects directly address this limitation while TB-500 enhances cellular migration to the injury site

Preferential accumulation at injury sites suggests active homing mechanisms or increased vascular permeability and tissue binding at sites of damage, though specific mechanisms governing injury-site localization remain under investigation

Macpherson AJ, Gatto D, Sainsbury E, Harriman GR, Hengartner H, Zinkernagel RM: A primitive T cell-independent mechanism of intestinal mucosal IgA responses to commensal bacteria

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