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doi: 10.3389/fnagi.2019.00204 35 ChesserASPritchardSMJohnsonGV
These dosages are derived from published preclinical studies and anecdotal reports
In addition, our in vitro studies indicate a novel mechanism, that is GSTP can be phosphorylated at Ser184 and enter the nucleus under LPS stimulation, and GSTP interacted with HMGB1 to inhibit cPKC-mediated phosphorylation of HMGB1 and association between HMGB1 and CRM1 in the nucleus
The one-carbon metabolism pathway highlights therapeutic targets for gastrointestinal cancer
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