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Abstract Tripeptide GSH is associated not only with the control and maintenance of redox cell homeostasis, but also with the processes of detoxification, proliferation, cell differentiation, and regulation of cell death
This specific property has become the focus of numerous research strategies, such as the development of senolytic agents like FOXO4-DRI, which selectively disrupt the FOXO4p53 interaction, thereby enabling the targeted elimination of dysfunctional cells
No synergistic adverse effects have been reported in clinical practice
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Confirmatory secondary endpoints included 20%, 25%, and 30% weight loss, with all analyses performed on an intention-to-treat basis and reported with 95% confidence intervals
