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The GLP-1R and GIPR pathways both converge on beta cell function through incretin signaling, and retatrutide provides a tool to study the amplified incretin response that occurs when both receptors are activated simultaneously

This is likely central to the healing acceleration seen but also raises theoretical concern: chronic pro-angiogenic stimulation in a context of occult malignancy could theoretically be problematic

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If weight changes occur, they're more plausibly secondary effectsthrough improved recovery, training consistency, or routine adherencerather than guaranteed fat loss