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This early stage mortality might be attributed to a rapid and acute physiological response
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PMID: Summary Keywords erectile dysfunction, immune-inflammatory interaction, molecular mechanisms, penile microenvironment, spermatogenic impairment, testicular microenvironment, varicocele Citation Xu G and Yu Q (2026) Advances in immune-inflammatory interactions within the testicular-penile microenvironment of varicocele patients: molecular mechanisms underlying erectile dysfunction and spermatogenic impairment
B-cell specific ablation of ChAT increases the mortality of mice subjected to partial hepatectomy compared to their wildtype counterpart, due to dysregulation of 7nAChR expressing Kupffer cells and CD8 + T cells, limiting their regenerative capacity and producing harmful IFN-, respectively (112)
