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J Trace Elem Med Biol 67:126801

Summary and perspectives This review highlights the pivotal role of amino acid metabolism in the pathogenesis of pulmonary fibrosis, with a focus on how dysregulated metabolic pathwaysparticularly those involving proline, arginine, glutamine, and tryptophandrive collagen deposition, fibroblast activation, and disease progression

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In murine ApoE(-/-) models, GHRP-2 was associated with elevated IGF-1 levels, reduced interferon-gamma, decreased superoxide production, and modulation of inflammatory gene expression