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Diabetes-associated excess kidney tissue RAS activation increases angiotensin II-mediated glomerular efferent arteriolar vasoconstriction and glomerular hyperfiltration, tubular sodium reabsorption, and signalling through intracellular pathways, resulting in inflammation and oxidative stress [13]
Patients experiences with GLP1-RAs a systematic review
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Prez-Fuentes N, Alvario R, Alfonso A, Gonzlez-Jartn J, Gegunde S, Vieytes MR, et al
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