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Cell 85 : 149158 Buschmann T, Potapova O, Bar-Shira A, Ivanov VN, Fuchs SY, Henderson S, Fried VA, Minamoto T, Alarcon-Vargas D, Pincus MR, Gaarde WA, Holbrook NJ, Shiloh Y and Ronai Z (2001) Jun NH2-terminal kinase phosphorylation of p53 on Thr-81 is important for p53 stabilization and transcriptional activities in response to stress

[130] reported that VEGF is a tumour growth factor, and that inhibiting VEGF activity decreased blood vessel density in tumours and suppressed tumour growth

Multiple organ failure including sepsis-induced cardiac dysfunction seem to be the result of numerous factors as overwhelming inflammation and nitric oxide synthesis impairment associated with mitochondrial dysfunction and increased oxidative stress (Cecchini and Cecchini, 2020)

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Hence, elucidation of the underlying mechanisms for cell premature senility may be a key to our understanding of liver fibrosis pathogenesis