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The phenolic hydroxyl groups in their molecular structure enable them to mimic the endogenous estrogen 17-estradiol by binding to estrogen receptors (ER and ER), thereby disrupting normal endocrine regulation

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After a single dose of FOXO4-DRI, more than half of the senescent cells disappeared

These effects are attributed to indirect modulation of signaling pathways involved in cell survival rather than direct stimulation of neuronal activity