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To delineate the functional role of PRMT7-mediated GLUD1 methylation in gastric cancer progression, we conducted CCK8 and EdU assays to assess cell proliferation, along with wound healing assays to evaluate migration

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Through EMT, epithelial cells lose cell polarity, connections to the basement membrane and other epithelial phenotypes, and gain mesenchymal phenotypes such as increased migratory and invasive abilities, anti-apoptotic features and the capacity to degrade the extracellular matrix (79)

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For patients with significant injury histories, the combination is almost always more appropriate than either peptide alone