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The study focused on alleviating symptoms rather than inhibiting tumor growth or destroying cancer cells, representing supportive care rather than anti-tumor activity
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The activation of HCA2 by -hydroxybutyrate in adipose tissue, therefore, serves as a negative feed-back loop on adipose tissue lipolysis by reducing the release of free fatty acids from adipocytes
Given that EtOH-induced mitochondrial dysfunction and ER stress are intimately involved in the pathogenesis of ALD, future studies to examine these pathways under the current experimental setting will provide important mechanistic information underlying the protection seen in GCLM KO mice
Time for a new metric for hypoxic dose
In the study of cell death interactions, the problem of mechanistic isolation is prominent
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