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These findings suggest that the OGC accounts for the low-affinity high capacity of GSH transport in liver mitochondria, and imply that the nature of the high affinity GSH transporter remains to be identified

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Rare reports of this lesion in rats appear in literature

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287 This theoretical perspective shifts the focus from static oncogenic drivers to the dynamic stability and plasticity of the signaling network as a whole, explaining why monotherapies often fail and providing a rationale for combinatorial strategies that target network vulnerabilities

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Marso, S

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