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Thymosin Beta-4 binds G-actin monomers and keeps them unpolymerised, creating the intracellular conditions fibroblasts and endothelial cells need to migrate toward damaged tissue

It takes a significantly longer time to build or accrue the benefit, and then half as long to destroy all of that benefit. For example, what took three years to build three years of cardiovascular benefit was effectively erased or undone in just one and a half years after stopping

More provocative is that the directionality of the variants effect reported in the Nature paper is the opposite of these previous studies

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