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Thus, modulating the Fc:FcRn interaction to prolong plasma half-life and attain a favorable biodistribution is a clinical success, and several clinical-stage candidate therapeutics harbor either the YTE or LS amino acid substitutions, or a more recently reported triple Fc mutant variant (Q311R/M428E/N434W

2012;96(3):62134, xi

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Heres how a new human proteome atlas could improve target selection, predict toxicity and support drug repurposing

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