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Dextran-coated iron oxide nanoparticles (DCIONs), upon magnetic field activation and at specific concentrations, could promote Y79 cell death by activating TNF- activity in Y97 cells through the caspase-3/7 pathway

Ligand binding drives receptor autophosphorylation, IRS-1/IRS-2 adapter recruitment, and activation of two primary downstream cascades: PI3K/Akt/mTOR (governing protein synthesis, glucose uptake, and cell survival) and MAPK/ERK (regulating cell proliferation and differentiation)

Laboratory research provides valuable insights into biological mechanisms, but findings from in vitro and preclinical studies do not automatically translate to human outcomes

The viability of the cells was calculated as a percentage of viability of the AP probe-net treated cells compared to the control

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