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Endothelial dysfunction due to eNOS uncoupling: molecular mechanisms as potential therapeutic targets

doi: 10.1038/s41593-021-00975-6 390 XieX.MaG.LiX.ZhaoJ.ZhaoZ.ZengJ.et al

While these radicals are essential for pathogen clearance, excessive or chronic inflammation can lead to tissue damage due to the persistent high levels of free radicals [58]

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During the activation phase, stimuli such as Nigericin (113) prompted NLRP3 to oligomerize through the NACHT structural domain, recruit apoptosis-associated speck-like protein containing a CARD (ASC) and NIMA-associated kinase 7 (NEK7), and promote ASC oligomerization to form macromolecular foci of specks, which in turn led to pro-caspase-1 recruitment and activation, and assembly to form NLRP3 inflammatory vesicles (114)