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Therefore, we hypothesize that BPC157 may reduce intracellular ROS levels through an antioxidant mechanism similar to that reported in J Clin Invest [19], thereby stabilizing BACH1 protein and enabling its pro-angiogenic function

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Disorganized collagen, neovascularization with abnormal nerve growth, and fatty infiltration characterize chronic tendinopathy

"BPC 157's effect on healing." Journal of Physiology-Paris 91.3-5 (1997): 173-178

Bradshaw WG, Baum MJ, Awh CC

Yik MY, Azlan A, Rajasegaran Y, Rosli A, Yusoff NM, Moses EJ (2021) Mechanism of human telomerase reverse transcriptase (hTERT) regulation and clinical impacts in leukemia