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Japan, in particular, reports a heightened prevalence of hereditary renal hypouricemia, with approximately 90% of Japanese hypouricemia patients attributable to non-functional variants of URAT1 [60]

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Signs of oxidative damage to proteins, lipids, and DNA have been found in blood [35, 112], urine [113], and post-mortem brain samples [79, 80] collected from autistic individuals