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revealed that DCBLD1, as an oncogene, can also undergo lactylation, and the lactylation of DCBLD1 can inhibit its ubiquitination, thereby stabilizing the DCBLD1 protein
Metalloproteinase regulation studies document MMP1 and MMP2 gene expression increases at 0.01 nM with TIMP1 increases at all tested concentrations (0.01-100 nM)
It functions by donating the hydrogen atom to fatty peroxyl radicals and disrupting the lipid peroxidation process
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All of these can deplete glutathione levels or interfere with its absorption
Furthermore, enhanced accumulation at the tumor site has been observed in DOX-containing liposomes or chitosan-based micelles after encapsulation within monocytes/macrophages [172, 173]
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