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Differential inhibition of prostaglandin endoperoxide synthase (cyclooxygenase) isozymes by aspirin and other non-steroidal anti-inflammatory drugs
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The biological mechanism underlying the rodent findings involves GLP-1 receptor stimulation on thyroid C-cells, which produce calcitonin
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Glucagon-like peptide receptor agonists and dipeptidyl peptidase-4 inhibitors in the treatment of diabetes: a review of clinical trials
