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Maiti A, MBBS, DiNardo CD, MD, MSCE, Rausch CR, PharmD, Pemmaraju N, MD, Garcia-Manero G, MD, Ohanian M, DO, Daver N, MD, Issa GC, MD, Borthakur G, MD, Ravandi F, MBBS, Alvarado Y, MD, Kadia TM, MD, Jabbour EJ, MD, Short NJ, MD, Montalban-Bravo G, MD, Jain N, MD, Kornblau SM, MD, Masarova L, MD, Wierda WG, Sasaki K, MD, PhD, Takahashi K, MD, PhD, Yilmaz M, MD, Burger JA, MD, PhD, Estrov ZE, MD, Verstovsek S, MD, PhD, Andreeff M , MD, PhD, Bose P, MD, Ferrajoli A, MD, Thompson PA, Montalbano K, Vaughan K, RN, Bivins CA, RN, Pierce SA, BSN, BA, Qiao W, PhD, Ning J, Welch JS, MD, PhD, Kantarjian H, MD, Konopleva M, MD, PhD

We previously demonstrated that melanocytes obtained from normally pigmented vitiligo skin show cellular and molecular alterations characterized by constitutive activation of antioxidant enzymes 1 and defects in mitochondrial metabolism reflected in altered expression and activity of complex I, increased generation of reactive oxygen species (ROS), low ATP production, and modified expression of some glycolytic enzymes 2

Next, 10 ng of each DNA mixture were tested by ddPCR using the appropriate combination of donor-specific and patient-specific reactions, selected in accordance with the screening results (Supplementary Fig

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The peptide, humanin, was ultimately traced to a tiny snippet of mitochondrial DNApart of the ninety-eight per cent of the human genome that had long been dismissed as junk DNA. Cohens work helped reveal that, in the three-billion-letter book that is our genome, even obscure one-liners can be an important part of the story