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The traditional classification of microglial phenotypes into binary M1 (pro-inflammatory) and M2 (anti-inflammatory/neuroprotective) categories has provided a useful conceptual framework, though it is increasingly recognized as an oversimplification of their functional diversity (Figure 5)
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Modulating EAAT2, AQP4, and purinergic signaling offers a unique opportunity for targeted intervention, selectively disrupting neurotoxic cascades while preserving the essential homeostatic functions of astrocytes
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