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GLP-1 has been demonstrated to induce natriuresis and diuresis, likely involving the inhibition of the sodiumhydrogen exchanger 3 (NHE3) localized at the brush border of the renal proximal tubular cells (192)
Indexing terms: Acquired Immunodeficiency Syndrome, diabetes mellitus, cardiovascular diseases, kidney diseases, carnitine, therapeutics
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It represents a second, complementary pathway through which microdosed GLP-1 could influence autoimmune activity
