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The resultant accumulation of labile iron intensifies oxidative stress via Fenton chemistry and ultimately renders dopaminergic neurons highly susceptible to ferroptosis (50)
This specific ICV dose was empirically derived from a systemic equivalent of 10 mg/kg, adjusted based on the approximate brain-to-body weight ratio of a mouse, approximately 1:100
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[1] [2] How DSIP Works: Mechanisms of Action DSIP exerts its effects through multiple mechanisms involving neurotransmitter modulation, hormonal regulation, and stress axis interactions
If iron ions serve as key mediators, strategies should target ferroportin and iron transport
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