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Disruption of this inhibitory interaction allows increased nitric oxide production, which is relevant to endothelial signaling and vascular responsiveness in experimental systems [3]

Extended-duration effects of GLP-1 RAs and multi-agonists on liver-associated outcomes involving cirrhosis development, HCC incidence, and death from hepatic causes are being elucidated through ongoing observational studies and long-term trial extensions [65-70]

Cammalleri, L

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