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While the significance of endothelial dysfunction in the onset of atherosclerosis is widely recognized, its specific contribution to the pathogenesis of CVD has yet to be definitively determined (Figure 3)
Studies in older mice investigating individual cells as well as larger vessels and micro-vessels will be useful in determining which cells of the cerebrovasculature are responsible for these changes and whether there are APOE -driven differences in FAO in both larger and smaller vessels that can affect transport functions
