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It must be used under medical supervision to be safe

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Archived from the original on September 16, 2016

Inactivation of S1PR2, for example, a sphingosine receptor which also binds to Nogo-A preventing axonal growth, resulted in increased remyelination in both lysolecithin-induced demyelination and EAE mice ( Figure 2 As myelin debris accumulate in the extracellular matrix and anomalous remyelination occurs, it has been suggested that demyelinating chronic disease may be associated with Nogo-A deposition and increased signaling

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