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The study confirmed that impairment of the GSH redox cycle but not inhibition of catalase dramatically increased susceptibility of pulmonary artery endothelial cells to H2O2-mediated attack

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Normal bone marrow function is suppressed, leading to anemia, thrombocytopenia, and leukopenia, manifesting in fatigue, bleeding, and/or recurrent infections (Kyle et al., 2003)

C., Hui, L., & Bianchi, D