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Existing findings indicate that the protective mechanism is mainly attributed to the GLP-1 signaling pathway, which involves cyclic adenosine monophosphate (cAMP), protein kinase A (PKA) and cAMP response element-binding protein (CREB)

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However, when the experimental conditions are designed to reflect patients with severe liver injury, lack of regeneration, and acute liver failure, a dose of 600 mg/kg APAP must be used [121]

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