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This is a general guideline, not a guarantee

To determine if GSH depletion-induced oxidative stress impairs mitochondrial biogenesis, we used MitoTracker Red to stain live mitochondria

U- 13 C5 l-proline and U- 13 C6 arginine were acquired from Cambridge Isotope Laboratory Inc

Emerging DDSs for gout and hyperuricemia, including zinc ferrite nanoparticles and synthetic gene networks, demonstrate innovative mechanisms such as self-regulated enzyme activity and biomimetic catalysis

Tracking clonal evolution during treatment in ovarian cancer using cell-free DNA

Eventually, GABA is removed from the system by converting it into succinate, which is then used in the Krebs cycle in the mitochondria to make ATP.[ref] The enzyme succinic semialdehyde dehydrogenase ( ALDH5A1 gene) is needed to convert GABA into succinate.[ref] Rare genetic mutations in the ALDH5A1 gene cause succinic semialdehyde dehydrogenase deficiency, which decreases the breakdown of GABA