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Expression of the tau protein and amyloid protein precursor processing genes in the CA3 area of the hippocampus in the ischemic model of Alzheimers disease in the rat

The violations fall into two categories: flawed written procedures and failure to investigate reportable events

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suggests that TB-500 may interact with cell movement as it binds globular actin (G-actin) and may modulate how actin filaments assemble to plausibly make endothelial cells more able to change shape, migrate, and form multicellular structures

However, treatments targeting amyloid have not yielded strong results in clinical trials, prompting scientists to seek alternative explanations and new therapeutic strategies for Alzheimers (Daly Timothy et al., 2021)