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Loss of DPEP1 markedly relieved FA- or cisplatin-induced AKI by altering the apoptosis and ferroptosis, but not the pyroptosis or necroptosis
When the skin is damaged, the process of hemolysis releases hemoglobin (a pro-oxidant), which can further contribute to the induction of HO-1 [124]
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illustrated how the selective permeability of the blood-brain-barrier (BBB) and bloodCSF barrier, combined with the spatial distribution of glutathione-metabolizing enzymes, underlies region-specific vulnerability to oxidative injury in the CNS (9)
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