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During the progression of CCl 4 -induced hepatic steatosis and fibrogenesis in vivo, CCl 4 induced gradually activation of SA--gal, a biomarker of cell senescence, along with a time-dependent elevation of oxidation in hepatocytes

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Binding sites were also localised to a minority of ovarian and prostate cancers, although GLP-1R binding sites were not detected in colorectal, lung, liver, stomach or pancreatic cancers [160, 161]

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