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These findings help to explain some aspects of TZPs clinical efficacy, but from this experiment, its impossible to discriminate whether the different effects of TZP are the result of GIPR signaling (85, 117, 118)

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10.1016/j.metabol.2013.10.005 115 MoraleM

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Theoretically, they operate through different mechanisms: semaglutide suppresses appetite and slows gastric emptying via GLP-1 receptors, while AOD 9604 acts on beta-3 adrenergic receptors on fat cells