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Published studies also examine S-glutathionylation, the reversible modification of protein cysteine residues that functions as a regulatory switch in redox signaling, and conjugation reactions catalyzed by the glutathione S-transferase enzyme family

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Sun 2021, PMID 34827486 a clean, independent APP/PS1 Alzheimer's-model mouse study)

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A complex network of the m6A-metabolite-TME axis has been proposed, i.e., the ability of m6A to induce aberrant changes in metabolite levels, which may in turn trigger oncogenic signaling pathways leading to significant alterations within the TME [43]

Presented for research literature review only Doedens L et al