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In contrast, the augmentation of IGF to rat follicles leads to an increase in both granulosa and theca cells [10]
Although basal XOR expression is typically low in humans, various factors such as hypoxia, ischemia-reperfusion injury, interleukin-1 (IL-1), interleukin-6 (IL-6), tumor necrosis factor-alpha (TNF-), and corticosteroid treatment can markedly enhance XOR transcription
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MSU crystals have been shown to trigger IL-1 production and inflammatory cytokine release through C5a activation, highlighting the potential of complement antagonists in managing gout inflammation
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