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Further structural analysis via the program AlphaFold 46 was conducted to visualize and better comprehend the interactions of the exendin 2029 sequence with TRPV1 within the broader context of GLP-1 and its derivatives (Supplementary Fig

Multiple cellular processes are considered to contribute to this functional decline and these are collectively referred to as the hallmarks of aging and these include processes like genomic instability, telomere attrition, epigenetic alterations, loss of proteostasis, disabled macroautophagy, deregulated nutrient-sensing, mitochondrial dysfunction, cellular senescence, stem cell exhaustion, altered intercellular communication, chronic inflammation, and dysbiosis 1

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