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HPG axis central effects: hypothalamus and pituitary Evidence on the potential neuroendocrine actions of GLP-1 on HPG axis was provided by an in vitro study in a rodent hypothalamic neuronal cell line (GT1-7) that reported a dose-dependent stimulating effect of GLP-1 on GnRH release, in association with a prompt increase in the plasma LH levels following the intracerebroventricular injection of GLP-1 in male rats [34]

This works regardless of the medication or concentration because the insulin syringe standardizes 100 units to 1 ml

This is likely because of the chronic lower oxygen supply to the brain [14, 15]

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Peptides like GHK-Cu and KPV inhibit NF-kB activation, reducing inflammatory markers by up to 60 percent in some studies

Reflections on the discovery GLP-1 as a satiety hormone: Implications for obesity therapy and future directions