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Curr Neuropharmacol 15:495518
1 2 American Family Physician
Oxidant damage in KashinBeck disease and a rat KashinBeck disease model by employing T2 toxin treatment under selenium deficient conditions
This extended urinary detection suggests continued metabolism of tissue-bound peptide or slow release from tissue reservoirs
It does not establish an approved therapeutic use or long-term safety profile
AT-SIRT3OE mice exhibited improved insulin sensitivity in HFD-fed mice, through alleviating infiltration of macrophage and pro-inflammatory macrophage polarization in the epididymal AT
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