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Oxidative stress and autophagy: The clash between damage and metabolic needs
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The mechanisms of action are fundamentally different: GLP-1 agonists are synthetic or modified peptides designed to mimic incretin hormones and directly stimulate specific cellular receptors, while allulose is a simple sugar that undergoes passive absorption and excretion
This observation may explain why semaglutide and other GLP-1 RAs ranked among the lowest-impact DM2 DMTs in our model compared to drugs such as metformin, which engage a broader range of metabolic and neuroprotective pathways (e.g., AMPK, insulin, and adipocytokine signaling)
Middle-aged and elderly adults are more prone to this condition, and it can occur at any age
That is roughly one in four people who take semaglutide and do not get the results they expected
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