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Notably, BPC157 accelerates burn wound healing after flame exposure [4], and our prior studies in thermal/incisional injury models implicate its proangiogenic activity as a central reparative mechanism [9]

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Researchers should consider whether a peptide mechanism of action could theoretically worsen the specific immune pathways that are already overactive in SLE

While individual hypersensitivity cannot be entirely eliminated, the clinical trial evidence is highly reassuring

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