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Extracellular deposition of -amyloid (A plaques), intraneuronal tau accumulation, inflammation (activated astrocytes and microglia), and neuronal loss are all consistent pathological features of the disease (Porquet et al., It is well established that A peptides existed in both fibrillary and non-fibrillar forms (Poduslo and Howell, 42 and A 40 are both neurotoxic using both human and murine neuronal cell cultures
10 The second clinical trial was a randomized, double-blind trial assessing the benefits of exenatide in 60 participants
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"A look at the R&D landscape in obesity, led by GLP-1s." August 2024
Longo et al., 2025)
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