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The lack of balance between the production of ROS and the antioxidant defense system of animals will lead to DNA hydroxylation, protein denaturation, lipid peroxidation, cell apoptosis, ultimately lead to cell damage (Yang et al., 2016)

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However, the actual metabolic changes are actually much more complicated: hyperthyroidism frequently causes moderate to severe endogenous insulin resistance, as demonstrated by high resting serum insulin concentrations and an exaggerated insulin response during an IV glucose tolerance test (8,9)

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