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doi:10.1016/s1096-7192(02)00037-9

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Eur J Obstet Gynecol Reprod Biol, 2005

We demonstrate mechanistically the need for increased mitochondrial function, facilitated by mTORPGC1-mediated mitochondrial biogenesis, to support rebalancing of pyrimidines via UCK2 to support cellular survival in the face of continued 5FU treatment