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The term Type III diabetes has been proposed to describe AD that may develop from glucose and insulin dysregulation at the CNS (213, 219)

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34,35 However, the controversy surrounding the possible benefits of GIPR agonism stems from two main observations: its impaired insulinotropic effects, 36,37 and the prevention of weight gain by suppression of endogenous GIP-induced adipogenesis, 38,39 However, these seemingly discouraging observations have been questioned by the finding of the central anorexic effects of GIP, 4042 and benefits of the GLP-1R/GIPR dual agonist LY3298176 (tirzepatide) in diabetic and overweight individuals

Following the study, the following was reported: (1) The peptide appeared to promote the outgrowth of tendon fibroblasts and tissue healing