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The ability of Tsp1a to reduce visceral hypersensitivity in a model of irritable bowel syndrome suggests that pharmacological inhibition of hNav1.7 at peripheral sensory nerve endings might be a viable approach for eliciting analgesia in patients suffering from chronic visceral pain, the researchers wrote.
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If side effects are severe at this dose, it may indicate sensitivity that warrants an even slower start through microdosing protocols
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